In the recent days, especially post Covid-19 waves, there were an increased risk of cardiovascular complications, including myocardial infarction (heart attack), in some individuals after recovering from COVID-19. Several factors may contribute to an increased risk of heart attacks in individuals post-COVID-19 although the research is prevailing towards this sudden spike in the condition.:
Inflammation: COVID-19 can lead to a systemic inflammatory response, and inflammation is a known risk factor for cardiovascular events. Inflammation may contribute to the formation of plaques in the arteries.
Endothelial Dysfunction: The virus can affect the endothelial cells lining blood vessels, leading to endothelial dysfunction. This dysfunction may contribute to the development of atherosclerosis (hardening and narrowing of the arteries) and increase the risk of heart attacks.
Hypercoagulability: COVID-19 has been associated with an increased risk of blood clot formation. This hypercoagulable state can lead to the formation of clots in the coronary arteries, resulting in a heart attack.
Direct Cardiac involvement: Some individuals with COVID-19 may experience direct damage to the heart muscle, known as myocarditis. This damage can weaken the heart and increase the risk of cardiovascular events.
Pre-existing Cardiovascular Conditions: Individuals with pre-existing cardiovascular conditions may be more susceptible to severe COVID-19, and the virus could exacerbate underlying cardiovascular issues, increasing the risk of heart attacks.